Impact of Tobacco Consumption on Cardiovascular and Respiratory Systems.
DOI:
https://doi.org/10.69980/ajpr.v28i5.634Keywords:
....Abstract
Tobacco consumption significantly harms both the cardiovascular and respiratory systems. It contributes to heart disease, high blood pressure, and stroke by damaging blood vessels and promoting atherosclerosis. In the lungs, tobacco smoke causes chronic inflammation, reduces lung function, and increases the risk of diseases like COPD and lung cancer. Reducing tobacco use through public health efforts is essential to prevent these life-threatening conditions.
Objective: Impact of Tobacco Consumption on Cardiovascular and Respiratory Systems
Methods: A cross-sectional study was conducted among voluntary male participants from Sarojini Naidu Medical College, Agra. The study included smokers aged 18–32 years with a minimum smoking history of one year.
Results: The study demonstrated significant cardiovascular and respiratory impairments associated with tobacco consumption. Among smokers, systolic and diastolic blood pressures were elevated compared to non-smokers, indicating increased cardiovascular strain. Pulmonary function tests revealed reduced lung capacity and airflow obstruction in smokers, with a marked decline in forced expiratory volume (FEV1) and forced vital capacity (FVC) compared to controls.
Conclusion: Tobacco consumption has a significant negative impact on both the cardiovascular and respiratory systems. It contributes to elevated blood pressure, increased cardiac workload, and impaired lung function, leading to a higher risk of heart disease, stroke, COPD, and lung cancer. Preventive measures and smoking cessation are essential to reduce these health risks and improve overall well-being.
References
1. World Health Organisation. Key facts on tobacco[Internet].2013.[Updated 2017 May; cited April 2013].Available from: http: // www.who.int/ mediacentre/ factsheets/ fs339/en.
2. Mishra S, Joseph RA, Gupta PC, Pezzack B, Ram F, Sinha DN, et al. Trends in bidi and cigarette smoking in India from 1998 to 2015 , by age , gender and education. BMJ Glob Heal. 2016;1:e000005:1 8.
3. United States Department of Health and Human Services. The Health consequences of Smoking - 50 Years of Progress A Report of the Surgeon General. A Rep Surg Gen. 2014;1081.
4. Ministry of Health & Family Welfare G of I. Tobacco taxes in India: An empirical Analysis. 2014.
5. Benowitz NL. Nicotine Addiction. N Engl J Med. 2010;362(24):2295 303.
6. Lewis A, Miller JH, Lea RA. Monoamine oxidase and tobacco dependence. Neurotoxicology. 2007;28(1):182 95.
7. Fowler JS, Logan J, Wang G-J, Volkow ND. Monoamine oxidase and cigarette smoking. Neurotoxicology.2003;24(1):75-82.
8. Jacob P, Benowitz NL, Copeland JR, Risner ME, Cone EJ. Disposition kinetics of nicotine and cotinine enantiomers in rabbits and beagle dogs. J Pharm Sci. 1988;77(5):396-400.
9. Benowitz NL, Jacob P, Herrera B. Nicotine intake and dose response when smoking reduced-nicotine content cigarettes.Clin Pharmacol Ther. 2006;80 (6):703 14.
10. Middlekauff HR, Park J, Moheimani RS. Adverse effects of cigarette and non cigarette smoke exposure on the autonomic nervous system: Mechanisms and implications for cardiovascular risk. J Am Coll Cardiol.2014;64(16):1740-50.
11. Catassi A, Servent D, Paleari L, Cesario A, Russo P. Multiple roles of nicotine on cell proliferation and inhibition of apoptosis: Implications on lung carcinogenesis. Mutat Res - Rev Mutat Res. 2008;659(3):221 31.
Downloads
Published
Issue
Section
License
Copyright (c) 2025 American Journal of Psychiatric Rehabilitation
This work is licensed under a Creative Commons Attribution 4.0 International License.
This is an Open Access article distributed under the terms of the Creative Commons Attribution 4.0 International License permitting all use, distribution, and reproduction in any medium, provided the work is properly cited.
